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DDAH1 Recombinant Rabbit Monoclonal Antibody [JM53-18] (ET1705-71)

Price: Regular price $355.00

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Catalog# ET1705-71

Rabbit Monoclonal to DDAH1

Tested for

  • WB

  • IHC-P

  • IP

React with

  • Human

Recombinant production enables lot-to-lot consistency and is animal-cruelty-free

Specifications

Product Type

Recombinant Rabbit monoclonal primary

Product Name

DDAH1 Recombinant Rabbit Monoclonal Antibody [JM53-18] (ET1705-71)

Immunogen

Recombinant protein within human ddah1 aa 100-285 / 285.

Host

Rabbit

Positive Control

Human kidney tissue lysate, human liver tissue lysate, human prostate tissue, human kidney tissue.

Conjugation

Unconjugated

Clonality

Monoclonal

Clone Number

JM53-18

APPLICATION DILUTION

  • WB

  • 1:500-1:2,000

  • IHC-P

  • 1:50-1:200

  • IP

  • Use at an assay dependent concentration.

PROPERTIES

Form

Liquid

Storage Condition

Store at +4C after thawing. Aliquot store at -20C or -80C. Avoid repeated freeze / thaw cycles.

Storage Buffer

1*TBS (pH7.4), 0.05% BSA, 40% Glycerol. Preservative: 0.05% Sodium Azide.

Concentration

1ug/ul

Purification

Protein A affinity purified.

Molecular Weight

Predicted band size: 31 kDa

Isotype

IgG

TARGET

UNIPROT #

O94760

PROTEIN NAME

DDAH1

SYNONYMS

DDAH antibody;DDAH I antibody;DDAH-1 antibody;DDAH1 antibody;DDAH1_HUMAN antibody;DDAHI antibody;Dimethylargininase 1 antibody;Dimethylargininase-1 antibody;Dimethylarginine dimethylaminohydrolase 1 antibody;N(G) antibody;N(G)-dimethylarginine dimethylaminohydrolase 1 antibody;NG NG dimethylarginine dimethylaminohydrolase antibody

SEQUENCE SIMILARITIES

Belongs to the DDAH family.

TISSUE SPECIFICITY

Detected in brain, liver, kidney and pancreas, and at low levels in skeletal muscle.

SUBCELLULAR LOCATION

Cytoplasm.

FUNCTION

DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production. Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation. DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues.

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